Browsing Department of Biology by Title
Research and Teaching Output of the MIT Community
- Home
- →
- Department of Biology
- →
- Browsing Department of Biology by Title
JavaScript is disabled for your browser. Some features of this site may not work without it.
Browsing Department of Biology by Title
-
(Massachusetts Institute of Technology, 2006)A collection of over 500 lines of zebrafish (Danio rerio), each heterozygous for a recessive embryonic lethal mutation caused by a retroviral insertion, was screened for lines that displayed early mortality and externally ...
-
(Massachusetts Institute of Technology, 1996)
-
(Massachusetts Institute of Technology, 2004)The heterodimerization of α- and β-tubulin represents a critical early step in microtubule morphogenesis. In vitro studies have defined a pathway that mediates the incorporation of monomeric tubulin polypeptides into ...
-
(Massachusetts Institute of Technology, 2008)Mitotic cyclin-dependent kinases (CDKs) are best known for their essential functions in triggering entry into M-phase, where they have established roles in nuclear envelope breakdown, chromosome condensation, and Golgi ...
-
(Massachusetts Institute of Technology, 2002)The process of programmed cell death is important in the development and homeostasis of multicellular organisms. The conserved morphological events of this process have been termed apoptosis. The molecular mechanisms of ...
-
(Massachusetts Institute of Technology, 2011)The class A and B synthetic multivulva (synMuv) genes of C. elegans redundantly antagonize an EGF/Ras pathway to prevent ectopic vulval induction. The class B synMuv genes encode many proteins known to remodel chromatin ...
-
(Massachusetts Institute of Technology, 2011)Most animals pass through a series of juvenile stages on their way from embryo to adult. These stages represent periods of time in which stage-specific developmental processes occur. At the end of development, the organism ...
-
(Massachusetts Institute of Technology, 2008)In the presence of a bacterial food source, the small nematode C. elegans greatly reduces its rate of locomotion. While mechanical agitation greatly stimulates the locomotion of well-fed animals on bacteria, it does not ...
-
(Massachusetts Institute of Technology, 1997)
-
(Massachusetts Institute of Technology, 1998)
-
(Massachusetts Institute of Technology, 2004)This suggests that the S phase cyclin, which first activates DNA replication, then acts locally at the origin to prevent pre-RC formation.
-
(Massachusetts Institute of Technology, 2012)The objective of this work is to obtain an in depth understanding of how embryonic stem cell-secreted signals contribute to their identity. We analyze the contribution of broad and specific signals present in the cell-secreted ...
-
(Massachusetts Institute of Technology, 2012)Regeneration of missing body parts is biologically fascinating, yet poorly understood. Many instances of regeneration, such as the replacement of amphibian limbs or planarian heads, require both a source for new cellular ...
-
(Massachusetts Institute of Technology, 1995)
-
(Massachusetts Institute of Technology, 2006)Huntington's disease (HD) is an autosomal dominant neurological disorder caused by a polyglutamine (polyQ) repeat expansion in the huntingtin (Htt) protein. The disease is characterized by neurodegeneration and formation ...
-
(Massachusetts Institute of Technology, 1994)
-
(Massachusetts Institute of Technology, 2004)Bacteria keep track of their population density by using extracellular signaling molecules. In response to the accumulation of these signals, changes in gene expression coordinate the switch from unicellular activity to ...
-
(Massachusetts Institute of Technology, 1987)
-
(Massachusetts Institute of Technology, 1997)
-
(Massachusetts Institute of Technology, 2011)Cataract, the leading cause of blindness worldwide, is the opacification of the eye lens. In.age-related cataract, as well as roughly one half of congenital cataract cases, aggregation or precipitation of crystallin proteins ...
Browse
My Account
MIT-Mirage