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dc.contributor.authorAshford, Nicholas
dc.contributor.authorBauman, Patricia
dc.contributor.authorBrown, Halina
dc.contributor.authorClapp, Richard
dc.contributor.authorFinkel, Adam
dc.contributor.authorGee, David
dc.contributor.authorHattis, Dale
dc.contributor.authorMartuzzi, Marco
dc.contributor.authorSasco, Annie
dc.contributor.authorSass, Jennifer
dc.date.accessioned2021-07-14T17:08:22Z
dc.date.available2021-07-14T17:08:22Z
dc.date.issued2015
dc.identifier.urihttps://hdl.handle.net/1721.1/131091
dc.descriptionLetter to the Editoren_US
dc.description.abstractIN THEIR REPORT “Variation in cancer risk among tissues can be explained by the number of stem cell divisions” (2 January, p. 78), C. Tomasetti and B. Vogelstein discuss an interesting correlation (0.804) between estimated lifetime stem cell division number in 31 tissue types and corresponding cancer incidence rates in the United States. However, their assertion that only 35% of cancer risk variation is due to environmental or genetic factors is problematic.en_US
dc.language.isoen_USen_US
dc.publisherScienceen_US
dc.rightsAttribution-NonCommercial-NoDerivs 3.0 United States*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/3.0/us/*
dc.titleCancer Risk: Role of Environmenten_US
dc.typeOtheren_US


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