| dc.contributor.author | Halme, Dina Gould | |
| dc.coverage.temporal | Spring 2004 | |
| dc.date.accessioned | 2023-03-06T17:34:52Z | |
| dc.date.available | 2023-03-06T17:34:52Z | |
| dc.date.issued | 2004-06 | |
| dc.identifier | 7.340-Spring2004 | |
| dc.identifier.other | 7.340 | |
| dc.identifier.other | IMSCP-MD5-aa7f926250a6625b662486dc7bd1efc9 | |
| dc.identifier.uri | https://hdl.handle.net/1721.1/148362 | |
| dc.description.abstract | Every infection consists of a battle between the invading pathogen and the resisting host. To be successful, a pathogen must escape the many defenses of the host immune system until it can replicate and spread to another host. A pathogen must prevent one of three stages of immune function: detection, activation, or effector function. Examples of disease-specific immune evasion and the mechanisms used by pathogens to prevail over their hosts' immune systems are discussed. Also considered is what these host-pathogen interactions reveal about the normal function of the immune system and basic cell biological processes, such as protein maturation and degradation. | en |
| dc.language.iso | en-US | |
| dc.rights | This site (c) Massachusetts Institute of Technology 2023. Content within individual courses is (c) by the individual authors unless otherwise noted. The Massachusetts Institute of Technology is providing this Work (as defined below) under the terms of this Creative Commons public license ("CCPL" or "license") unless otherwise noted. The Work is protected by copyright and/or other applicable law. Any use of the work other than as authorized under this license is prohibited. By exercising any of the rights to the Work provided here, You (as defined below) accept and agree to be bound by the terms of this license. The Licensor, the Massachusetts Institute of Technology, grants You the rights contained here in consideration of Your acceptance of such terms and conditions. | en |
| dc.rights | Attribution-NonCommercial-ShareAlike 3.0 Unported | * |
| dc.rights.uri | http://creativecommons.org/licenses/by-nc-sa/3.0/ | * |
| dc.subject | immunology | en |
| dc.subject | immune system | en |
| dc.subject | immune evasion | en |
| dc.subject | pathogen | en |
| dc.subject | effector function | en |
| dc.subject | infections | en |
| dc.subject | Human cytomegalovirus | en |
| dc.subject | Human Immunodeficiency Virus | en |
| dc.subject | CD4 cells | en |
| dc.subject | CD8 cells | en |
| dc.subject | T cells | en |
| dc.subject | surace receptors | en |
| dc.subject | cell lysis | en |
| dc.subject | host-pathogen interactions | en |
| dc.subject | host surveillance | en |
| dc.subject | antibodies | en |
| dc.subject | MHC class I | en |
| dc.subject | blood-borne pathogens | en |
| dc.subject | macrophages | en |
| dc.subject | phagocytosis | en |
| dc.subject | endocytosis | en |
| dc.subject | degradation | en |
| dc.subject | antigen | en |
| dc.subject | apoptosis | en |
| dc.subject | cytokines | en |
| dc.subject | immune response | en |
| dc.title | 7.340 Immune Evasion: How Sneaky Pathogens Avoid Host Surveillance, Spring 2004 | en |
| dc.title.alternative | Immune Evasion: How Sneaky Pathogens Avoid Host Surveillance | en |
| dc.audience.educationlevel | Undergraduate | |
| dc.subject.cip | 260507 | en |
| dc.subject.cip | Immunology | en |
| dc.date.updated | 2023-03-06T17:34:58Z | |
