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7.341 The DNA Damage Response as a Target for Anti-Cancer Therapy, Fall 2008

dc.contributor.authorReinhardt, Hans Christian
dc.coverage.temporalFall 2008
dc.date.accessioned2023-03-06T17:29:55Z
dc.date.available2023-03-06T17:29:55Z
dc.date.issued2008-12
dc.identifier7.341-Fall2008
dc.identifier.other7.341
dc.identifier.otherIMSCP-MD5-81ab5efd1184a04f9b9fca12b2b7e700
dc.identifier.urihttps://hdl.handle.net/1721.1/148358
dc.description.abstractCellular responses to DNA damage constitute one of the most important fields in cancer biology. In this class we will analyze classical and recent papers from the primary research literature to gain a profound understand of cell cycle regulation and DNA damage checkpoints that act as powerful emergency brakes to prevent cancer. This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. Many instructors of the Advanced Undergraduate Seminars are postdoctoral scientists with a strong interest in teaching.en
dc.language.isoen-US
dc.relation.isbasedonhttp://hdl.handle.net/1721.1/49423
dc.rightsThis site (c) Massachusetts Institute of Technology 2023. Content within individual courses is (c) by the individual authors unless otherwise noted. The Massachusetts Institute of Technology is providing this Work (as defined below) under the terms of this Creative Commons public license ("CCPL" or "license") unless otherwise noted. The Work is protected by copyright and/or other applicable law. Any use of the work other than as authorized under this license is prohibited. By exercising any of the rights to the Work provided here, You (as defined below) accept and agree to be bound by the terms of this license. The Licensor, the Massachusetts Institute of Technology, grants You the rights contained here in consideration of Your acceptance of such terms and conditions.en
dc.rightsAttribution-NonCommercial-ShareAlike 3.0 Unported*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/3.0/*
dc.subjectDNAen
dc.subjectdamage checkpointsen
dc.subjectcanceren
dc.subjectcellsen
dc.subjecthuman cellsen
dc.subjectexogenousen
dc.subjectendogenousen
dc.subjectcheckpointsen
dc.subjectgeneen
dc.subjectsignalingen
dc.subjectcancer biologyen
dc.subjectcancer preventionen
dc.subjectprimary sourcesen
dc.subjectdiscussionen
dc.subjectDNA damageen
dc.subjectmolecularen
dc.subjectenzymeen
dc.subjectcell cycleen
dc.subjectextracellular cuesen
dc.subjectgrowth factorsen
dc.subjectCdk regulationen
dc.subjectcyclin-dependent kinaseen
dc.subjectp53en
dc.subjecttumor suppressoren
dc.subjectapoptosisen
dc.subjectsignalingen
dc.subjectMDC1en
dc.subjectH2AXen
dc.subjectRad50en
dc.subjectFluorescence activated cell sorteren
dc.subjectChk1en
dc.subjectmutanten
dc.title7.341 The DNA Damage Response as a Target for Anti-Cancer Therapy, Fall 2008en
dc.title.alternativeThe DNA Damage Response as a Target for Anti-Cancer Therapyen
dc.typeLearning Object
dc.contributor.departmentMassachusetts Institute of Technology. Department of Biology
dc.audience.educationlevelUndergraduate
dc.subject.cip260911en
dc.subject.cipOncology and Cancer Biologyen
dc.date.updated2023-03-06T17:30:01Z


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