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dc.contributor.authorBell, Stephen P
dc.date.accessioned2017-06-14T13:20:19Z
dc.date.available2017-06-14T13:20:19Z
dc.date.issued2017-01
dc.identifier.issn2050-084X
dc.identifier.urihttp://hdl.handle.net/1721.1/109848
dc.description.abstractHuman cells that lack a subunit in their origin recognition complex are viable, which suggests the existence of alternative mechanisms to initiate DNA replication.en_US
dc.language.isoen_US
dc.publishereLife Sciences Publications, Ltd.en_US
dc.relation.isversionofhttp://dx.doi.org/10.7554/eLife.24052en_US
dc.rightsCreative Commons Attribution 4.0 International Licenseen_US
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/en_US
dc.sourceeLifeen_US
dc.titleRethinking origin licensingen_US
dc.typeArticleen_US
dc.identifier.citationBell, Stephen P. “Rethinking Origin Licensing.” eLife 6 (January 2017): e24052 © 2017 Bellen_US
dc.contributor.departmentMassachusetts Institute of Technology. Department of Biologyen_US
dc.contributor.mitauthorBell, Stephen P
dc.relation.journaleLifeen_US
dc.eprint.versionFinal published versionen_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dspace.orderedauthorsBell, Stephen Pen_US
dspace.embargo.termsNen_US
dc.identifier.orcidhttps://orcid.org/0000-0002-2876-610X
mit.licensePUBLISHER_CCen_US


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