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dc.contributor.authorChan, Suk-tak
dc.contributor.authorKwong, Kenneth K
dc.date.accessioned2017-03-28T15:56:42Z
dc.date.available2017-03-28T15:56:42Z
dc.date.issued2016-12
dc.date.submitted2016-10
dc.identifier.issn1178-2021
dc.identifier.urihttp://hdl.handle.net/1721.1/107754
dc.description.abstractWe read with interest the article “Cortical spreading depression produces a neuroprotective effect activating mitochondrial uncoupling protein-5” published in Neuropsychiatr Dis Treat by Viggiano et al.1 The authors showed that cerebral spreading depression (CSD) triggered uncoupling protein-5 (UCP-5),1 which had been reported to exert a long-term effect upon neuron protection.2 The result is another piece in CSD literature on modifying gene expressions to provide neuroprotection to subsequent ischemic episodes.3,4en_US
dc.language.isoen_US
dc.publisherDove Medical Pressen_US
dc.relation.isversionofhttp://dx.doi.org/10.2147/ndt.s125677en_US
dc.rightsCreative Commons Attribution-NonCommercial 3.0 Unporteden_US
dc.rights.urihttps://creativecommons.org/licenses/by-nc/3.0/en_US
dc.sourceDove Medical Pressen_US
dc.titleNeuroprotection and acidosis induced by cortical spreading depressionen_US
dc.typeArticleen_US
dc.identifier.citationKwong, Kenneth, and Suk-tak Chan. “Neuroprotection and Acidosis Induced by Cortical Spreading Depression.” Neuropsychiatric Disease and Treatment Volume 12 (2016): 3191–3194.en_US
dc.contributor.departmentHarvard University--MIT Division of Health Sciences and Technologyen_US
dc.contributor.mitauthorKwong, Kenneth K
dc.relation.journalNeuropsychiatric Disease and Treatmenten_US
dc.eprint.versionFinal published versionen_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dspace.orderedauthorsKwong, Kenneth; Chan, Suk-taken_US
dspace.embargo.termsNen_US
mit.licensePUBLISHER_CCen_US


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