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dc.contributor.authorYe, Xin
dc.contributor.authorWeinberg, Robert A
dc.date.accessioned2018-07-13T13:15:31Z
dc.date.available2018-07-13T13:15:31Z
dc.date.issued2017-11
dc.date.submitted2017-11
dc.identifier.issn1949-2553
dc.identifier.urihttp://hdl.handle.net/1721.1/116966
dc.description.abstractThe cell-biological program termed epithelial-mesenchymal transition (EMT) plays a key role in adenocarcinoma progression, invasion, and metastasis. An EMT operating within carcinoma cells can be activated by a variety of paracrine signals arising in the tumor microenvironment. These EMT-inducing signals trigger profound transcription changes through activation of EMT-inducing transcription factors (EMT-TFs), leading to repression of genes associated with the epithelial differentiation program and activation of those associated with the mesenchymal program.en_US
dc.publisherImpact Journals, LLCen_US
dc.relation.isversionofhttp://dx.doi.org/10.18632/ONCOTARGET.22432en_US
dc.rightsCreative Commons Attribution 3.0 Unported licenseen_US
dc.rights.urihttp://creativecommons.org/licenses/by/3.0/en_US
dc.sourceOncotargeten_US
dc.titleThe SUMO guards for SNAILen_US
dc.typeArticleen_US
dc.identifier.citationYe, Xin, and Robert A. Weinberg. “The SUMO Guards for SNAIL.” Oncotarget 8, no. 58 (November 15, 2017).en_US
dc.contributor.departmentMassachusetts Institute of Technology. Department of Biologyen_US
dc.contributor.mitauthorWeinberg, Robert A
dc.relation.journalOncotargeten_US
dc.eprint.versionFinal published versionen_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dc.date.updated2018-07-12T14:34:51Z
dspace.orderedauthorsYe, Xin; Weinberg, Robert A.en_US
dspace.embargo.termsNen_US
dc.identifier.orcidhttps://orcid.org/0000-0002-0895-3557
mit.licensePUBLISHER_CCen_US


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