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dc.contributor.authorBlanchard, Joel W.
dc.contributor.authorTsai, Li-Huei
dc.contributor.authorBlanchard, Joel W.
dc.date.accessioned2019-11-15T16:56:27Z
dc.date.available2019-11-15T16:56:27Z
dc.date.issued2019-03
dc.identifier.issn1934-5909
dc.identifier.urihttps://hdl.handle.net/1721.1/122952
dc.description.abstractConflicting clinical studies have reported that statins both reduce and accelerate cognitive impairments in Alzheimer’s disease. In this issue, Van der Kant et al. (2019) use iPSC-derived neurons to thoroughly dissect the link between cholesterol synthesis, phospho-Tau, and amyloid-β, revealing new therapeutic opportunities in Alzheimer’s disease and related dementias.en_US
dc.publisherElsevier BVen_US
dc.relation.isversionofhttp://dx.doi.org/10.1016/j.stem.2019.02.003en_US
dc.rightsCreative Commons Attribution-NonCommercial-NoDerivs Licenseen_US
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/en_US
dc.sourceProf. Tsai via Courtney Crummetten_US
dc.titleUnraveling the Paradox of Statins with Human Neurons: New Leads in Alzheimer’s Diseaseen_US
dc.typeArticleen_US
dc.identifier.citationBlanchard, Joel W. and Tsai, Li-Huei. "Unraveling the Paradox of Statins with Human Neurons: New Leads in Alzheimer’s Disease." Cell Stem Cell 24, 3 (March 2019): 347-349 © 2019 Elsevieren_US
dc.contributor.departmentPicower Institute for Learning and Memoryen_US
dc.relation.journalCell Stem Cellen_US
dc.eprint.versionAuthor's final manuscripten_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dspace.date.submission2019-11-04T20:20:43Z
mit.journal.volume24en_US
mit.journal.issue3en_US
mit.licenseOPEN_ACCESS_POLICY


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