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dc.contributor.authorHorton, Brendan
dc.contributor.authorSpranger, Stefani
dc.date.accessioned2020-04-15T16:20:36Z
dc.date.available2020-04-15T16:20:36Z
dc.date.issued2018-07
dc.identifier.issn1074-7613
dc.identifier.urihttps://hdl.handle.net/1721.1/124657
dc.description.abstractPancreatic adenocarcinoma (PDAC) is mostly refractory to immunotherapies. In this issue of Immunity, Li et al. (2018) generate a library of clonal PDAC tumors to examine the tumor-intrinsic features shaping the anti-tumor immune response and find that tumor cell-derived CXCL1 directly blunts T cell infiltration and reduces responsiveness to immunotherapy.en_US
dc.language.isoen
dc.publisherElsevier BVen_US
dc.relation.isversionof10.1016/j.immuni.2018.07.001en_US
dc.rightsCreative Commons Attribution-NonCommercial-NoDerivs Licenseen_US
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/en_US
dc.sourcePMCen_US
dc.subjectImmunologyen_US
dc.subjectImmunology and Allergyen_US
dc.subjectInfectious Diseasesen_US
dc.titleA Tumor Cell-Intrinsic Yin-Yang Determining Immune Evasionen_US
dc.typeArticleen_US
dc.identifier.citationHorton, Brendan and Stefani Spranger. "A Tumor Cell-Intrinsic Yin-Yang Determining Immune Evasion." Immunity 49 (2018): 11-13 © 2018 The Author(s)en_US
dc.contributor.departmentMassachusetts Institute of Technology. Department of Biologyen_US
dc.contributor.departmentKoch Institute for Integrative Cancer Research at MITen_US
dc.relation.journalImmunityen_US
dc.eprint.versionFinal published versionen_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dc.date.updated2020-01-30T17:55:11Z
dspace.date.submission2020-01-30T17:55:13Z
mit.journal.volume49en_US
mit.journal.issue1en_US
mit.licensePUBLISHER_CC
mit.metadata.statusComplete


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