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dc.contributor.authorUhler, Caroline
dc.contributor.authorShivashankar, G. V.
dc.date.accessioned2021-03-11T21:33:33Z
dc.date.available2021-03-11T21:33:33Z
dc.date.issued2020-04
dc.identifier.issn1471-0072
dc.identifier.issn1471-0080
dc.identifier.urihttps://hdl.handle.net/1721.1/130124
dc.description.abstractCoronaviruses employ various nuclear signalling mechanisms for their propagation. Here we link replication of coronaviruses, including SARS-CoV-2, the coronavirus disease 2019 (COVID-19)-causing virus, with cytoskeleton-dependent signalling and 3D genome organization. This link could provide novel insights into the high coronavirus pathogenicity in certain cellular states, particularly those associated with ageing.en_US
dc.publisherSpringer Science and Business Media LLCen_US
dc.relation.isversionofhttps://doi.org/10.1038/s41580-020-0242-zen_US
dc.rightsCreative Commons Attribution-Noncommercial-Share Alikeen_US
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/4.0/en_US
dc.sourceProf. Uhler via Phoebe Ayersen_US
dc.titleMechano-genomic regulation of coronaviruses and its interplay with ageingen_US
dc.typeArticleen_US
dc.identifier.citationUhler, Caroline and G. V. Shivashankar. "Mechano-genomic regulation of coronaviruses and its interplay with ageing." Nature Reviews Molecular Cell Biology 21, 5 (April 2020): 247–248. © 2020 Springer Nature Limiteden_US
dc.contributor.departmentMassachusetts Institute of Technology. Laboratory for Information and Decision Systemsen_US
dc.relation.journalNature Reviews Molecular Cell Biologyen_US
dc.eprint.versionAuthor's final manuscripten_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dspace.date.submission2021-03-05T17:23:36Z
mit.journal.volume21en_US
mit.journal.issue5en_US
mit.licenseOPEN_ACCESS_POLICY
mit.metadata.statusComplete


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