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dc.contributor.authorLeRoux, Michele
dc.contributor.authorCulviner, Peter H
dc.contributor.authorLiu, Yue J
dc.contributor.authorLittlehale, Megan L
dc.contributor.authorLaub, Michael T
dc.date.accessioned2021-10-27T20:22:31Z
dc.date.available2021-10-27T20:22:31Z
dc.date.issued2020
dc.identifier.urihttps://hdl.handle.net/1721.1/135215
dc.description.abstract© 2020 Elsevier Inc. The toxin-antitoxin (TA) systems encoded on bacterial chromosomes have long been thought to act as effectors of a cellular stress response system. LeRoux et al. find that, despite strong transcriptional induction following various stress treatments, the toxins of TA systems are not activated under these conditions.
dc.language.isoen
dc.publisherElsevier BV
dc.relation.isversionof10.1016/J.MOLCEL.2020.05.028
dc.rightsCreative Commons Attribution-NonCommercial-NoDerivs License
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/
dc.sourcePMC
dc.titleStress Can Induce Transcription of Toxin-Antitoxin Systems without Activating Toxin
dc.typeArticle
dc.contributor.departmentMassachusetts Institute of Technology. Department of Biology
dc.contributor.departmentHoward Hughes Medical Institute
dc.relation.journalMolecular Cell
dc.eprint.versionAuthor's final manuscript
dc.type.urihttp://purl.org/eprint/type/JournalArticle
eprint.statushttp://purl.org/eprint/status/PeerReviewed
dc.date.updated2021-07-21T14:12:55Z
dspace.orderedauthorsLeRoux, M; Culviner, PH; Liu, YJ; Littlehale, ML; Laub, MT
dspace.date.submission2021-07-21T14:12:57Z
mit.journal.volume79
mit.journal.issue2
mit.licensePUBLISHER_CC
mit.metadata.statusAuthority Work and Publication Information Needed


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