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dc.contributor.authorSidorov, Michael Samuel
dc.contributor.authorAuerbach, Benjamin David
dc.contributor.authorBear, Mark
dc.date.accessioned2013-04-30T19:40:57Z
dc.date.available2013-04-30T19:40:57Z
dc.date.issued2013-04
dc.date.submitted2013-01
dc.identifier.issn1756-6606
dc.identifier.urihttp://hdl.handle.net/1721.1/78638
dc.description.abstractLoss of the translational repressor FMRP causes Fragile X syndrome. In healthy neurons, FMRP modulates the local translation of numerous synaptic proteins. Synthesis of these proteins is required for the maintenance and regulation of long-lasting changes in synaptic strength. In this role as a translational inhibitor, FMRP exerts profound effects on synaptic plasticity.en_US
dc.publisherBioMed Central Ltden_US
dc.relation.isversionofhttp://dx.doi.org/10.1186/1756-6606-6-15en_US
dc.rightsCreative Commons Attributionen_US
dc.rights.urihttp://creativecommons.org/licenses/by/2.0en_US
dc.sourceBioMed Central Ltden_US
dc.titleFragile X mental retardation protein and synaptic plasticityen_US
dc.typeArticleen_US
dc.identifier.citationSidorov, Michael S, Benjamin D Auerbach, and Mark F Bear. “Fragile X Mental Retardation Protein and Synaptic Plasticity.” Molecular Brain 6.1 (2013): 15.en_US
dc.contributor.departmentMassachusetts Institute of Technology. Department of Brain and Cognitive Sciencesen_US
dc.contributor.departmentPicower Institute for Learning and Memoryen_US
dc.contributor.mitauthorSidorov, Michael Samuel
dc.contributor.mitauthorAuerbach, Benjamin David
dc.contributor.mitauthorBear, Mark
dc.relation.journalMolecular Brainen_US
dc.eprint.versionFinal published versionen_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dc.date.updated2013-04-25T23:06:09Z
dc.language.rfc3066en
dc.rights.holderMichael S Sidorov et al.; licensee BioMed Central Ltd.
dspace.orderedauthorsSidorov, Michael S; Auerbach, Benjamin D; Bear, Mark Fen
mit.licensePUBLISHER_CCen_US
mit.metadata.statusComplete


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