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dc.contributor.authorLamming, Dudley W.
dc.contributor.authorSabatini, David M.
dc.contributor.authorSabatini, David
dc.date.accessioned2014-12-16T17:33:55Z
dc.date.available2014-12-16T17:33:55Z
dc.date.issued2011-06
dc.identifier.issn15504131
dc.identifier.urihttp://hdl.handle.net/1721.1/92330
dc.description.abstractTOR (target of rapamycin) signaling regulates life span in many organisms, but the mechanism behind the effect is unknown. In this issue of Cell Metabolism, Pan and colleagues (2011) find that reduced TORC1 activity promotes yeast life span via a mechanism that, paradoxically, relies upon the production of normally deleterious reactive oxygen species.en_US
dc.language.isoen_US
dc.publisherElsevieren_US
dc.relation.isversionofhttp://dx.doi.org/10.1016/j.cmet.2011.05.006en_US
dc.rightsArticle is made available in accordance with the publisher's policy and may be subject to US copyright law. Please refer to the publisher's site for terms of use.en_US
dc.sourceElsevieren_US
dc.titleA Radical Role for TOR in Longevityen_US
dc.typeArticleen_US
dc.identifier.citationLamming, Dudley W., and David M. Sabatini. “A Radical Role for TOR in Longevity.” Cell Metabolism 13, no. 6 (June 2011): 617–618. © 2011 Elsevier Inc.en_US
dc.contributor.departmentMassachusetts Institute of Technology. Department of Biologyen_US
dc.contributor.departmentWhitehead Institute for Biomedical Researchen_US
dc.contributor.departmentKoch Institute for Integrative Cancer Research at MITen_US
dc.contributor.mitauthorSabatini, David M.en_US
dc.relation.journalCell Metabolismen_US
dc.eprint.versionFinal published versionen_US
dc.type.urihttp://purl.org/eprint/type/JournalArticleen_US
eprint.statushttp://purl.org/eprint/status/PeerRevieweden_US
dspace.orderedauthorsLamming, Dudley W.; Sabatini, David M.en_US
dc.identifier.orcidhttps://orcid.org/0000-0002-1446-7256
mit.licensePUBLISHER_POLICYen_US
mit.metadata.statusComplete


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